| 释义 |
Definition of beta-adrenergic in US English: beta-adrenergicadjective Relating to or affecting beta receptors. sympathetic nerves that stimulate beta-adrenergic receptors Example sentencesExamples - The researchers' earlier experiments showed that disrupting the function of PI K preserves beta-adrenergic receptors on heart cells when they are chronically exposed to adrenaline and thus preserves heart function.
- Drugs that impair detrusor contractility and increase urethral tone include calcium channel blockers, anticholinergics, alpha- and beta-adrenergic agonists, narcotics, antidepressants, and antipsychotics.
- The present understanding of Anolis chromatophore control suggests that losers have either exhausted EPI needed to change the ratio of alpha- and beta-adrenergic receptor stimulation or that MSH may prevail.
- Patients taking beta-adrenergic blockers present a special challenge because beta blockade may limit the effectiveness of epinephrine.
- This effect was blocked by a monoclonal antibody to interleukin - 10, but not by antagonists to transforming growth factor-beta 1, prostaglandins, or beta-adrenergic agonists.
- Over time, these over-excited receptors become desensitized to the effects of norepinephrine, largely due to the effects of a second molecule, beta-adrenergic receptor kinase.
- In congestive heart failure, the beta-adrenergic receptor system fails to work properly.
- Melatonin is a hormone with multiple functions in humans, produced by the pineal gland and stimulated by beta-adrenergic receptors.
- Stimulation of beta-adrenergic receptors on mast cells inhibits the secretion of inflammatory mediators.
- Patients taking certain medications, including alpha- and beta-adrenergic blocking agents, oxytocic medications, sympathomimetic agents, general anesthetics, and monoamine oxidase inhibitors are more sensitive.
- Some patients with life-threatening asthma do not respond to inhaled selective beta-adrenergic agents and subsequently advance to respiratory failure.
- Recommendations for the use of beta-adrenergic blockers in the setting of acute myocardial infarction include the addition of a Class I recommendation for use of these agents in patients with non-ST-segment elevation infarctions.
- Drug therapy includes aspirin, beta-adrenergic blockers, cholesterol-lowering agents and other anti-ischemic drugs that can ameliorate angina and improve the patient's quality of life.
- For beta 1 selectivity, a beta-adrenergic antagonist should contain a p-substituted aryloxypropanolamine pharmacophore.
- These include vesnarinone, beta-adrenergic receptor blockers, and calcium channel blocking agents.
- Beta blockers are also known as beta-adrenergic blocking agents.
- Stimulation of beta-adrenergic receptors results in a moderate increase in chloride secretion in isolated opercular membranes from seawater-adapted fish, and is associated with an increase in cAMP.
- The cardiac actions of beta-adrenergic stimulation are not affected by iNO.
- The use of beta-adrenergic blocking agents in the treatment of heart failure and left ventricular dysfunction has provided a significant improvement in survival and reduced the need for hospitalization for worsening heart failure.
- The effect may depend predominately on the blockade of central beta-adrenergic receptors.
Definition of beta-adrenergic in US English: beta-adrenergicadjective Relating to or affecting beta receptors. sympathetic nerves that stimulate beta-adrenergic receptors Example sentencesExamples - Stimulation of beta-adrenergic receptors results in a moderate increase in chloride secretion in isolated opercular membranes from seawater-adapted fish, and is associated with an increase in cAMP.
- The researchers' earlier experiments showed that disrupting the function of PI K preserves beta-adrenergic receptors on heart cells when they are chronically exposed to adrenaline and thus preserves heart function.
- Melatonin is a hormone with multiple functions in humans, produced by the pineal gland and stimulated by beta-adrenergic receptors.
- For beta 1 selectivity, a beta-adrenergic antagonist should contain a p-substituted aryloxypropanolamine pharmacophore.
- The use of beta-adrenergic blocking agents in the treatment of heart failure and left ventricular dysfunction has provided a significant improvement in survival and reduced the need for hospitalization for worsening heart failure.
- Drugs that impair detrusor contractility and increase urethral tone include calcium channel blockers, anticholinergics, alpha- and beta-adrenergic agonists, narcotics, antidepressants, and antipsychotics.
- Patients taking certain medications, including alpha- and beta-adrenergic blocking agents, oxytocic medications, sympathomimetic agents, general anesthetics, and monoamine oxidase inhibitors are more sensitive.
- The present understanding of Anolis chromatophore control suggests that losers have either exhausted EPI needed to change the ratio of alpha- and beta-adrenergic receptor stimulation or that MSH may prevail.
- These include vesnarinone, beta-adrenergic receptor blockers, and calcium channel blocking agents.
- This effect was blocked by a monoclonal antibody to interleukin - 10, but not by antagonists to transforming growth factor-beta 1, prostaglandins, or beta-adrenergic agonists.
- In congestive heart failure, the beta-adrenergic receptor system fails to work properly.
- Recommendations for the use of beta-adrenergic blockers in the setting of acute myocardial infarction include the addition of a Class I recommendation for use of these agents in patients with non-ST-segment elevation infarctions.
- Drug therapy includes aspirin, beta-adrenergic blockers, cholesterol-lowering agents and other anti-ischemic drugs that can ameliorate angina and improve the patient's quality of life.
- Over time, these over-excited receptors become desensitized to the effects of norepinephrine, largely due to the effects of a second molecule, beta-adrenergic receptor kinase.
- Stimulation of beta-adrenergic receptors on mast cells inhibits the secretion of inflammatory mediators.
- The cardiac actions of beta-adrenergic stimulation are not affected by iNO.
- Some patients with life-threatening asthma do not respond to inhaled selective beta-adrenergic agents and subsequently advance to respiratory failure.
- Beta blockers are also known as beta-adrenergic blocking agents.
- The effect may depend predominately on the blockade of central beta-adrenergic receptors.
- Patients taking beta-adrenergic blockers present a special challenge because beta blockade may limit the effectiveness of epinephrine.
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